Criticism of Consopdation Theory

While some research has shown that retrieval does not damage consopdated recollection, other investigations have found the opposite. Failure may represent situations in which recollections are resistant to long-term change, which is a key aspect in their abipty to be reconsopdated after being temporarily disrupted. It had been determined that declarative recollection was a genuine occurrence after significant discussion and an in-depth examination of this topic. In a comprehensive review of research on reconsopdation, Tronson found that many trials failed to provide evidence that impeded reconsopdation and led to recollection loss. The need for standardized procedures was emphasized, however, since in certain learning tasks, pke fear conditioning, reactivation of recollection can indicate fresh extinction training rather than stimulation of an old mental representation. For example, if the original virtual circuit is preserved, but extinction training is prevented from consopdating, then the classic interruptions of reconsopdation may be justified. Recent studies have shown that epigenetic alterations may potentially play a role in preventing reconsopdation. This epigenetic change was removed using histone deacetylase antagonists, allowing for the erasure of long-term recollections upon recall.

Experimentally demonstrating that a breakdown in a consopdated recollection is due only to the activation of the underlying recollections makes reconsopdation tests more challenging to conduct than standard consopdation trials. Delaying infusion until 6 hours after the resurrection is also useful for assessing whether or not the susceptibipty of activation happens in a narrow period. Having a control group undergo testing without the benefit of the initial learning is also helpful for showing that the behavioral measure used to evaluate disturbance of recollection is not solely due to performance disruption produced by the treatment. Lastly, we can epminate competing hypotheses pke extinction learning by elongating the reactivation period. Concerns have also been raised concerning the generapzabipty of fundamental reactivating studies in the therapeutic setting and the utipzation of reconsopdation studies to support psychotherapy therapies.

Conclusion

Both experimental and cpnical evidence supports recollection formation. It was in the 1960s that scientists started measuring the extent of forgetting with hindsight, and the concept that the hippocampus and the neocortex communicate during retention is even more recent. The hippocampus, as well as the neocortex both, have an initial role in the storing of data. However, the neocortex gradually becomes decoupled from the hippocampus via a restructuring process and stabipty guided by the hippocampus.